Thelansis

 Community-acquired pneumonia (CAP) denotes an acute infection affecting the pulmonary parenchyma in individuals devoid of recent hospitalization within 14 days and not residing in nursing homes or long-term care facilities. The etiology of CAP encompasses two broad categories: Typical agents (e.g., Streptococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, Staphylococcus aureus, Group A Streptococci, anaerobes, and gram-negative organisms) and Atypical agents (e.g., Legionella, Mycoplasma, Chlamydia pneumoniae, and C. psittaci), with H. influenzae and S. pneumoniae commonly implicated as bacterial causative agents. Pharyngeal colonization by pathogens, followed by micro-aspiration, constitutes the primary mechanism of lower respiratory tract invasion, supplemented by hematogenous dissemination and macro-aspiration. Pneumonia manifests when host pulmonary defenses are compromised or overwhelmed by a substantial pathogen inoculum or virulence. Risk factors for CAP enco...

 Community-Acquired Pneumonia (CAP) is an acute pulmonary parenchymal infection in an individual who has not been hospitalized in the last 14-days and is not a resident of a nursing home or long-term care facility. The pathogen causing Community-acquired pneumonia is classified into; Typical agents (Streptococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, Staphylococcus aureus, Group A Streptococci, anaerobes, and gram-negative organisms), and Atypical agents (Legionella, Mycoplasma, Chlamydia pneumoniae, and C. psittaci) globally. H. influenzae and S. pneumoniae are the two most common causes of bacterial pneumonia. Colonization of the pharynx with pathogens, followed by micro-aspiration, is the mechanism of entry into the lower respiratory tract. Other mechanisms include hematogenous spread and macro-aspiration. In the interaction between the pathogen and host pulmonary defence, pneumonia develops if the host defence is weak or overcome by high inoculum or virulen...